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Overexpression of PERK enhanced eIF2α phosphorylation and induced ATF4, BSP and OCN expression, showing The PERK-eIF2α-ATF4 signaling pathway regulates osteoblastic differentiation in response to endoplasmic reticulum stress (ERS)

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Notably, cell death of erythrocytes cannot be triggered by staurosporine [23], which is a potent apoptosis-inducing agent, but it is triggered by the Ca 2+ ionophore ionomycin [79]